000 -LEADER |
fixed length control field |
04426cam a2200553 i 4500 |
001 - CONTROL NUMBER |
control field |
17856518 |
005 - DATE AND TIME OF LATEST TRANSACTION |
control field |
20170913120551.0 |
008 - FIXED-LENGTH DATA ELEMENTS--GENERAL INFORMATION |
fixed length control field |
130819s2013 nyua g b 001 0 eng c |
010 ## - LIBRARY OF CONGRESS CONTROL NUMBER |
LC control number |
2013948228 |
016 7# - NATIONAL BIBLIOGRAPHIC AGENCY CONTROL NUMBER |
Record control number |
101624940 |
Source |
DNLM |
020 ## - INTERNATIONAL STANDARD BOOK NUMBER |
International Standard Book Number |
9781461483120 (alk. paper) |
020 ## - INTERNATIONAL STANDARD BOOK NUMBER |
International Standard Book Number |
1461483123 (alk. paper) |
020 ## - INTERNATIONAL STANDARD BOOK NUMBER |
Canceled/invalid ISBN |
9781461483137 (eBook) |
035 ## - SYSTEM CONTROL NUMBER |
System control number |
(OCoLC)ocn847348501 |
041 ## - LANGUAGE CODE |
Language code of text/sound track or separate title |
eng |
042 ## - AUTHENTICATION CODE |
Authentication code |
pcc |
050 00 - LIBRARY OF CONGRESS CALL NUMBER |
Classification number |
QP363.2 |
Item number |
.N53 2013 |
060 00 - NATIONAL LIBRARY OF MEDICINE CALL NUMBER |
Classification number |
2014 B-075 |
060 10 - NATIONAL LIBRARY OF MEDICINE CALL NUMBER |
Classification number |
WL 102 |
082 04 - DEWEY DECIMAL CLASSIFICATION NUMBER |
Classification number |
612.81045 |
Edition number |
23 |
Item number |
S N |
100 1# - MAIN ENTRY--PERSONAL NAME |
Personal name |
Suzumura, Akio |
9 (RLIN) |
1454 |
245 10 - TITLE STATEMENT |
Title |
Neuron-glia interaction in neuroinflammation / |
Statement of responsibility, etc. |
Akio Suzumura, Kazuhiro Ikenaka, editors. |
260 ## - PUBLICATION, DISTRIBUTION, ETC. (IMPRINT) |
Place of publication, distribution, etc. |
new york : |
Name of publisher, distributor, etc. |
springer sience + business media ; |
Date of publication, distribution, etc. |
2013 . |
265 ## - SOURCE FOR ACQUISITION/SUBSCRIPTION ADDRESS [OBSOLETE] |
Source for acquisition/subscription address [OBSOLETE] |
اقرأ للنشر و التوزيع |
300 ## - PHYSICAL DESCRIPTION |
Extent |
x, 187 pages : |
Other physical details |
illustrations (some color) ; |
Dimensions |
24 cm. |
490 1# - SERIES STATEMENT |
Series statement |
Advances in neurobiology, |
International Standard Serial Number |
2190-5215 ; |
Volume/sequential designation |
volume 7 |
504 ## - BIBLIOGRAPHY, ETC. NOTE |
Bibliography, etc |
Includes bibliographical references and index. |
505 00 - FORMATTED CONTENTS NOTE |
Title |
Acute, Chronic, and Nonclassical Neuroinflammation: Definitions in a Changing Scientific Environment / |
Statement of responsibility |
Robert P. Lisak, Joyce A. Benjamin -- |
Title |
Neuroinflammation in Neurological Disorders / |
Statement of responsibility |
Jun-ichi Kira -- |
Title |
Factors from Intact and Damaged Neurons / |
Statement of responsibility |
Tetsuya Mizuno -- |
Title |
Interactions Between Neurons and Microglia During Neuroinflammation / |
Statement of responsibility |
Akio Suzumura -- |
Title |
Neuron-Astrocyte Interactions in Neuroinflammation / |
Statement of responsibility |
Jasna Kriz -- |
Title |
Neuron-Oligodendrocyte Interactions in Neuroinflammation / |
Statement of responsibility |
Kazuhiro Ikenaka -- |
Title |
Neuron-Glia Interaction via Neurotrophins / |
Statement of responsibility |
Cinthia Farina -- |
Title |
Glial Communication via Gap Junction in Neuroinflammation / |
Statement of responsibility |
Hideyuki Takeuchi -- |
Title |
Toll-Like Receptors and Neuroinflammation / |
Statement of responsibility |
Sung Joong Lee -- |
Title |
The Blood-Brain Barrier in Neuroinflammation / |
Statement of responsibility |
Fumitaka Shimizu. |
520 ## - SUMMARY, ETC. |
Summary, etc. |
Accumulation of glia, gliosis, in various neurological disorders is not a static scar, but actively involved in pathogenesis of various neurological and psychiatric disorders, where glial cells produce both inflammatory and neurotrophic factors. These factors may play a role in neuronal damage, but also have a protective and reparative function by inducing neuroinflammation. However, definition as well as the mechanisms of neuroinflammation is not yet clear. We first define acute, chronic and non-classical neuroinflammation. Glial cells are activated by a variety of stimuli via receptors on glial cells. Toll like receptors (TLR) are one of these receptors. In response to harmful stimuli, neurons produce factors as either eat-me or help-me signals. These factors include cytokines, chemokines and damage-associated molecular pattern (DAMP). Some of them activate glial cells via TLR, and function to protect neurons or further induce neuroinflammation. Thus, the interaction between neuron-glia and glia-glia is a main feature of neuroinflammation. Glial cells communicate with other glial or neural cells via gap-junctions. The communication may also be important for the understanding of neuroinflammation. Oligodendrocytes-neurons communication may be critical in either myelination or demyelination. Damage of blood-brain barrier (BBB) is common feature of both inflammatory and degenerative neurological disorders. Thus, relation of BBB damage and functions of glial cell may also be important in the development of neuroinflammation. In this book, we focused on neuron-glia interaction of various aspects for understanding of pathophysiology of neuroinflammation in development of inflammatory as well as degenerative neurological disorders. |
650 14 - SUBJECT ADDED ENTRY--TOPICAL TERM |
Topical term or geographic name as entry element |
Neuroglia. |
9 (RLIN) |
1456 |
650 14 - SUBJECT ADDED ENTRY--TOPICAL TERM |
Topical term or geographic name as entry element |
Neural transmission. |
9 (RLIN) |
1457 |
650 #0 - SUBJECT ADDED ENTRY--TOPICAL TERM |
Topical term or geographic name as entry element |
Nervous system |
General subdivision |
Degeneration. |
9 (RLIN) |
1458 |
650 #0 - SUBJECT ADDED ENTRY--TOPICAL TERM |
Topical term or geographic name as entry element |
Inflammation. |
9 (RLIN) |
1459 |
650 12 - SUBJECT ADDED ENTRY--TOPICAL TERM |
Topical term or geographic name as entry element |
Neuroglia |
General subdivision |
physiology. |
650 22 - SUBJECT ADDED ENTRY--TOPICAL TERM |
Topical term or geographic name as entry element |
Inflammation |
General subdivision |
physiopathology. |
650 22 - SUBJECT ADDED ENTRY--TOPICAL TERM |
Topical term or geographic name as entry element |
Nervous System Physiological Phenomena. |
650 22 - SUBJECT ADDED ENTRY--TOPICAL TERM |
Topical term or geographic name as entry element |
Neurodegenerative Diseases. |
650 22 - SUBJECT ADDED ENTRY--TOPICAL TERM |
Topical term or geographic name as entry element |
Neuroimmunomodulation. |
650 #7 - SUBJECT ADDED ENTRY--TOPICAL TERM |
Topical term or geographic name as entry element |
Inflammation. |
Source of heading or term |
fast |
650 #7 - SUBJECT ADDED ENTRY--TOPICAL TERM |
Topical term or geographic name as entry element |
Nervous system |
General subdivision |
Degeneration. |
Source of heading or term |
fast |
650 #7 - SUBJECT ADDED ENTRY--TOPICAL TERM |
Topical term or geographic name as entry element |
Neural transmission. |
Source of heading or term |
fast |
650 #7 - SUBJECT ADDED ENTRY--TOPICAL TERM |
Topical term or geographic name as entry element |
Neuroglia. |
Source of heading or term |
fast |
700 1# - ADDED ENTRY--PERSONAL NAME |
Personal name |
Suzumura, Akio, |
Relator term |
editor. |
700 1# - ADDED ENTRY--PERSONAL NAME |
Personal name |
Ikenaka, Kazuhiro, |
Relator term |
editor. |
830 #0 - SERIES ADDED ENTRY--UNIFORM TITLE |
Uniform title |
Advances in neurobiology ; |
Volume number/sequential designation |
v. 7. |
942 ## - ADDED ENTRY ELEMENTS (KOHA) |
Source of classification or shelving scheme |
|
Koha item type |
Books |
Item part |
Faculty of Engineering كلية الهندسة |